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Additionally, research has illuminated potential mechanisms involved in TGF-1-induced ASM remodeling, encompassing pathways such as the phosphorylation of p38 and ERK1/2 (284), the up-regulation of miR-181a and miR-328-3p through the suppression of phosphatase and tensin homolog (PTEN) protein and increased p-Akt/Akt ratio suggesting TGF-1 induced ASM remodeling involves PTEN and Akt signaling pathway (285, 286), and the modulation of intracellular calcium levels [(Ca2+)i] and/or activation of Rho-associated protein kinase (ROCK) (287)

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The effect of acupuncture on oxidative stress: A systematic review and meta-analysis of animal models

FIGURE 2 3.1 Genetic factors Genetic components play a pivotal role in the pathophysiology of CD

Compared with normal hepatic function, the estimated ratio of the mean AUC 0 was 0.99 (0.891.11) in mild impairment, 1.01 (0.911.12) in moderate impairment and 1.11 (0.961.30) in severe impairment