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FIN56 is an another classic ferroptosis inducer, where the graphdiyne nanoplatforms GDY-FIN56-RAP effectively promotes FIN56-mediated selective lysosomal degradation of GPX4, exhibiting potential anti-glioma efficacy[121]

(1998) c-Src kinase activity is required for hepatocyte growth factor-induced motility and anchorage-independent growth of mammary carcinoma cells

Macrophages, platelets, and mesenchymal stem cells contribute to the EMT at primary sites, allowing for tumor cells to separate from neighboring epithelial cell-to-cell contacts and acquire an invasive phenotype

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Various grades of carbopol provided different In Vitro release pattern