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Accordingly, we obtained two data matrices: one containing metabolite concentrations for each lipedema patient (LP) and the other containing metabolite concentrations for each healthy control (HC) subject

Furthermore, variants in COL3A1 , such as the p.Gly637Ser mutation, have been linked to collagen synthesis abnormalities in a single family with the autosomal dominant hEDS phenotype that was identified which led to reduced collagen secretion and the over-modification of collagen [5,73]