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Thus, patients with sarcoidosis exhibited a greater than 8-fold increase in circulating ILC1s

Additional support for the model of GST regulation is provided by the observations that either immunodepletion of GST , or its inhibition by a rationally designed GSH-based-peptidomimetic inhibitor, - -glutamyl- S -(benzyl)cysteinyl- R ()-phenyl glycine diethyl ester (TLK199), also results in the activation of JNK