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When the riboflavin lack was corrected, some of the early morphologic and cell motility alterations in the gastrointestinal tract were observed in weanling rats given a riboflavin-deficient diet until weaning was irreversible (109, 110)

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If methaemoglobin induction accounts for all, or part, of the antidotal action of sodium nitrite, this might explain the potentiation noted with concomitant oxygen administration (Mansouri, 1985), although other investigators have not reached this conclusion (Way et al., 1966)

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