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Subsequent research revealed that GHK has exceptionally high binding affinity for copper ions (Cu), forming a stable chelate complex (GHK-Cu) that is the biologically active form responsible for the peptide's remarkable effects

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Mounting evidence suggests that m 6 A writers are upregulated in cancer, leading to dysregulation of m 6 A modification to promote tumorigenesis, malignant progression, and poor prognosis [26, 27]