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Rapid reduction of MDCK cell cholesterol by methyl--cyclodextrin alters steady state transepithelial electrical resistance

When ROS accumulation exceeds the tipping point, their carcinogenic effects on proliferation and development are shifted to antitumour effects via the induction of programmed cell death (PCD) [47, 49], including apoptosis, autophagy, necroptosis, pyroptosis, and ferroptosis

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The functions of glutathione peroxidase in ROS homeostasis and fruiting body development in Hypsizygus marmoreus

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