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In conditions with low H 2 S signaling (such as heart failure with reduced ejection fraction), SQOR inhibition may be beneficial to restore H 2 S levels, whereas in conditions primarily involving mitochondrial dysfunction (such as ischemiareperfusion injury), SQOR activation may be advantageous to support mitochondrial function

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In the context of HF, idebenone has been shown to inhibit myocardial lipid peroxidation, thereby contributing to cardiac protection

Janus kinase 2 modulates the apolipoprotein interactions with ABCA1 required for removing cellular cholesterol

1B) and an increase in the myotube diameters (Fig