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Abstract Intestinal microbiota metabolism of choline and phosphatidylcholine produces trimethylamine (TMA), which is further metabolized to a proatherogenic species, trimethylamine- N -oxide (TMAO)

These findings combined revealed a previously undescribed signaling of the PRMT1-UBE2m-NEDD4-PPAR axis that plays an essential role in fatty acid metabolism during CaOx crystals-induced kidney injury

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doi: 10.1093/nar/gkx343 5 BrownL.VillegasJ

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