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(2004) Proceedings of the National Academy of Sciences (PNAS) , 101(39): 1490714912 Foundational study identifying the EPO/-common receptor heteromer (IRR) as the mediator of EPOs tissue-protective effects, distinct from the EPOR homodimer responsible for erythropoiesis

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This breakdown or dyshomeostasis of biometals whereby antioxidant protection is overwhelmed in favor of elevated concentration of metals leads to a state of neurotoxicity with damaging interaction to proteins, lipid peroxidation, and DNA, all evidence of increased oxidative stress in the AD affected brain (Jomova et al., 2010

In another in vitro and in vivo study, Nrf2/HO-1 was also demonstrated to play an important inhibitory role in the pro-fibrotic pathway activated by TGF-1, which was verified by oligo-fucoidan in NRK-52E cells and kidney tissues of C57BL/6 mice [151]