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Ghafouri-Fard S, Balaei N, Shoorei H, Hasan SMF, Hussen BM, Talebi SF, Taheri M, Ayatollahi SA

The pathogenesis involves volume expansion from impaired sodium excretion and inappropriate activation of the renin-angiotensin-aldosterone system due to localized renal ischemia

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For many it is hormone correction, metabolic work, or a GLP-1 medication, with peptides as an adjunct or not at all

Unfortunately, many SARMs are so far stalled in phase II clinical trials limiting their large-scale implementation or trials are no longer recruiting patients due to the lack of results