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NIX/BNIP3L has been shown to be involved in the PINK1-parkin pathway, being ubiquitinated by parkin and recruiting the mitochondrial phagocytosis receptor, NBR1, for mitochondrial degradation [17]

Abnormalities in the mitochondrial electron transport chain (ETC), combined with impaired antioxidant defenses, lead to excessive accumulation of reactive oxygen species (ROS), which damage lipids, proteins, and mitochondrial DNA, ultimately promoting tubular apoptosis, interstitial fibrosis, and glomerulosclerosis ( Vitamin D mitigates oxidative stress and mitochondrial damage through multiple mechanisms

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Given BPC-157s gastric origins and gut-healing properties, minor changes in digestion may occur