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Description

Archived (PDF) from the original on 26 January 2017

10.1126/science.1248373 10 DuK

Our results are in line with those of Schurr et al

78 Inhibition of ferroptosis can also result from low glucose availability in the TME, which triggers AMP-activated protein kinase (AMPK)-mediated phosphorylation of acetyl-CoA carboxylases and inhibits production of fatty acids, including PUFAs that are necessary for ferroptosis