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M.LewinskiA.Karbownik-LewinskaM.KopchickJ

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doi: 10.1176/appi.ajp.2020.20091382 14

reported that the anti-HGF mAb ficlatuzumab significantly inhibited the c-MET activation and rescued the capacity of cetuximab to inhibit the phosphorylation of EGFR in cetuximab-resistant CRC cell lines LM1215, SW48, and DiFi ( Rilotumumab , a fully human anti-HGF mAb that neutralizes HGF-dependent c-MET signaling, has been investigated in phase Ib/II trial (NCT00788957) in combination with panitumumab in previously treated patients with KRAS-WT mCRC