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Some work on the hypothalamus

The bivariate distribution of amyloid- and tau: relationship with established neurocognitive clinical syndromes

p53 activates G1 checkpoint following DNA damage by doxorubicin during transient mitotic arrest

Our data show a further increase in fibroblasts proliferation sustained by a more energetic state, maintained throughout both the early and the late stage of the disease

Overall, the metabolomics results of these published cerebrospinal fluid investigations of a wide range of disease conditions did not match our ME/CFS cerebrospinal fluid pattern of disordered serine pathways with sarcosine and phospholipid production, folate and one carbon exchange, purine, pyrimidine, aromatic and anaplerotic amino acid metabolism