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As mentioned above, erastin or sorafenib treatment can activate Nrf2 via the P62-keap1 pathway, enabling the activation of a series of target genes capable of eliciting detoxification and antioxidation response, which contributes to ferroptosis resistance [7]

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10.1016/s0092-8674(03)00929-2 13 IsogaiS.MorimotoD.AritaK.UnzaiS.TennoT.HasegawaJ.et al (2011)

These changes reflect response to damage caused by high levels of homocysteine