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This knockdown also induces lactylation at the K73 site in ADP-ribosylation factor 1 (ARF1), which affects vesicle assembly and induces mitochondrial loading abnormalities, thereby blocking the release of mitochondria from astrocytes, ultimately exacerbating the extent of brain infarction in mice

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In normal cells, its antioxidant properties dominate because it directly reacts with superoxide anion radicals (O) and hydroxyl radicals (OH), which reduces the level of ROS within the cells (99)

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*Correspondence: Liming Shen [email protected] This article was submitted to Autism, a section of the journal Frontiers in Psychiatry Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers