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Description

Klotho deficiency is an early biomarker of renal ischemia-reperfusion injury and its replacement is protective

Such research should be actively pursued in the future

Targeting distinct pathogenetic pathways that drive disease progression has reached the stage of active drug development programs for disease modification with drugs targeting -synuclein, GBA-1, LRRK-2 and GLP1 receptors comprising the largest group of recent and ongoing disease modification trials in PD