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Among them, NHEJ reassembles a broken DNA end under DNA-PKcs, while HR is a high fidelity repair mechanism activated by ATM activation (Williams et al., 2007

Profound cellular defects attribute to muscular pathogenesis in the rhesus monkey model of Duchenne muscular dystrophy

MOTS-c treatment significantly reduced bone loss in 8-week-old C57BL/6 females mice with ovariectomies by suppressing osteoclast formation in an AMPK-dependent way [103]