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unraveled the function of GSH in keratinocytes and showed that GSH deficiency affects keratinocyte survival and wound repair

Importantly, we also found a significant downregulation of VLDLR and CPT1A, and upregulation of -SMA in human retinal sections from nAMD patients compared to age-matched non-AMD controls, particularly in the RPE layer (Fig

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This phenomenon, known as reperfusion injury, involves the oxidation of cellular components, mitochondrial dysfunction, and the activation of inflammatory pathways [91, 92]

Patients were dichotomised into two groups based on their transfer status and matched