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Acute fractures benefit from early peptide intervention (within days of injury) supporting rapid callus formation and preventing non-union complications

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More precisely, among them, the cyclin-dependent kinase inhibitor p21 Cip1/WAF1 , induced by p53 in response to OS and involved in the regulation of cell cycle arrest, replication, cellular proliferation, and apoptosis, competes with KEAP1 for the binding to NRF2 through the DLG motif, thus preventing NRF2 ubiquitination, and increasing its stabilization [184, 185]

Chemical aspects of UV-induced cross-linking of proteins to nucleic acids

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