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PCSK9i treatment inhibits platelet activation by modulating ox-LDL production and ox-LDL-mediated signaling pathway ( via improving the activity of antioxidants such as glutathione peroxidase, superoxide dismutase, and catalase, thereby counteracting lipid peroxidation, the production of ox-LDL as well as cell damage (Zhang et al., 2015

N-Acetylcysteine (NAC): A precursor to glutathione synthesis

Paraquat-induced apoptosis in human neuroblastoma SH-SY5Y cells: involvement of p53 and mitochondria