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Impaired methionine metabolism is a major contributor to hepatic GSH dysfunction in liver disease patients, and rodents naturally express higher levels of betaine-homocysteine methyltransferase, an enzyme which maintains proper hepatic methionine function [86]

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BMSCs inhibit pro-inflammatory cytokines, such as TNF- and IL-1, through the secretion of prostaglandin E2, indoleamine-2,3-dioxygenase, and TGF-