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On the mechanistic level [6], investigations have expanded beyond traditional core pathways such as OS, mitochondrial dysfunction, and neuroinflammation to encompass complex networks involving multi-organ system interactions like the gut-brain axis and spleen-brain axis

These findings led us to explore the consequences of PUFA exposure on IECs with impaired GPX4 activity in the intestine

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9,10 Maintaining a systolic blood pressure of 140 mm Hg or lower can decrease the development of diabetic peripheral neuropathy

This activation triggers pulsatile GH secretion while maintaining upstream hypothalamic regulation