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Residues E259 and Q252 are highlighted in red

372 Ongoing studies are evaluating ipilimumab in combination with TMZ, bevacizumab, and other ICIs

CD4 + CD25 + Foxp3 + Tregs cell dysfunction is common in autoimmune diseases (199), and it may be converted into pathogenic Th17 cells after repeated amplification (200)

This progressive neurodegenerative disorder is characterized by distinct pathological features, including the accumulation of iron in the substantia nigra pars compacta (SNc) and the aggregation of -syn within Lewy bodies, ultimately leading to the degeneration of dopaminergic neurons [2, 3]

BBD 1991 contains a sulfide quinone oxidoreductase gene for oxidation of sulfide, suggesting a mechanism for tolerating the sulfidic conditions of BBD mats