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Notably, mitochondria maintain a distinct pool of GSH that is independent of cytosolic GCL activity

Therefore, this review aims to provide a comprehensive summary of FRG-based prognostic models, their associated metabolic and signaling pathways, and promising therapeutic options for ferroptosis induction in NSCLC

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The Hcy values from supernatant and protein hydrolyzed fractions are added up to obtain tHcy