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ROS generation contributes to the activation of MLKL during necroptosis in lung cancer cells [131] and serves as a downstream event triggered by MLKL upon the induction of necroptosis in colon adenocarcinoma cells [132]

As previously reported, apoptotic PS exposure is mediated by caspase-3-activated XKR8 (ref

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The INK-ATTAC is a genetic tool based on dimerization of active caspase 8 domain by the AP20187 compound for inducible elimination of senescent cells under the control of p16Ink4a promoter

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