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N-acetylcysteine antagonizes NGF activation of TrkA through disulfide bridge interaction, an effect which may contribute to its analgesic activity

This is shown by the lines connecting the two groups and pointing toward the center of the sectors

Fine-Gray cumulative incidence analysis demonstrated a stepwise increase in CNS relapse risk, patients with at least two upregulated genes had the highest relapse rates, while those with none had the lowest ( Figure 5H )