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However, when liver damage progresses to non-alcoholic steatohepatitis (NASH) or cirrhosis, injured liver cells may release stored B12 into the bloodstream, causing elevated serum levels

BPC-157 has accumulated one of the largest and most diverse pre-clinical research profiles of any regenerative peptide: Gastrointestinal research originating from the University of Zagreb and subsequently replicated by other institutions has consistently documented BPC-157s protective and regenerative effects on gut mucosa in multiple ulcer and inflammatory bowel models establishing a strong mechanistic foundation for its continued use in digestive biology research

Instead, they recommended methylcobalamin, a form of B12 where a methyl group replaces the cyano group

No human liver-disease trial was identified

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