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This phosphatase inhibition leads to excess secretion of unusually large von Willebrand factor (ULVWF) multimers from vascular endothelial cells, and the anchorage of these long strings of ULVWF to endothelium leads to platelet adhesion and aggregation

Its relevance here comes from a 2018 murine study in which intranasal Semax at 0.5 mg/kg daily decreased LPS-induced serum TNF- and IL-1 by 45 to 50% and restored tight junction protein expression (claudin-2, occludin) over a 14-day intestinal inflammation model

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Mangalam, A

In parallel, depletion of mtDNA in cortical neurons (Lassmann et al., 2012)