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Of note, the myotendinous junction, as the weakest element in the muscle-tendon unit, its anatomy, and structural particularities were largely reviewed i.e., [1,2,23]

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Notably, the lipid profile of HSL-inhibited M2 macrophages shifted away from the M2 cluster and toward the M1 cluster, providing direct evidence that HSL activity is essential for maintaining the M2-specific lipidome (Supplementary Fig