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In the absence of serine hydroxymethyl transferase 2 (SHMT2), which catalyzes the first step of mitochondrial one-carbon metabolism, T cell abundance is diminished after antigen-specific activation, due to decreased nucleotide and glutathione synthesis (51, 67)

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& Perfetto, M

In conclusion, we show that APAP toxicity in HepG2 cells is largely independent of oxidative stress, and is linked instead to a decoupling of glycolysis from the TCA cycle, lactic acidosis, reduced NADPH production, and subsequent suppression of the anabolic pathways required for rapid growth

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