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Catalpol not only inhibits the over-proliferation of astrocytes in the ischemic cortex and DG, preventing neuroinflammation and scar formation from affecting the migration of newborn neurons, but also promotes recovery in the ischemic brain area by enhancing activation, providing neurotrophic substances, and supporting neuron survival, thus creating a favorable microenvironment for the migration of newborn neurons to the ischemic cortex area (Sun, 2022

5d), but also partially rescued the starvation-induced mortality in daf-12(rh61rh411) and din-1(dh127) mutants (Fig

A study showed FH -knockout ( FH /- ) cells are sensitive to ferroptosis, and such sensitivity is attributed to dysfunctional GPX4 (Kerins et al., 2018)

PMID: 8914118

The study compared the values of TAC and TOS in these patients with those of very low-risk PTC patients who were not assigned for RAI treatment