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In addition, experimental studies have shown that the elevation of GSH can inhibit the activation of hepatic stellate cells [33], which is a key driving factor for extracellular matrix deposition and fibrosis, thereby preventing the progression of liver fibrosis [34]

HIV-1 Tat-induced microglial activation via miRNA-34a-NF-B pathway The NF-B system, encompassing canonical and non-canonical branches, mediates inflammatory gene induction