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[DOI] [PMC free article] [PubMed] [Google Scholar] 249.Shelton MD

Lilley, J

6 AssuncaoM.AndradeJ

In a murine model of albumin-overload kidney disease, tubular cell-specific knockdown of RTN1A blunted ER stress and apoptosis of tubular cells, and reduced kidney fibrosis ( These findings are of relevance from a translational perspective since, besides 4-phenylbutyric acid, additional drugs approved for clinical use, albeit not for chronic kidney disease (CKD), are able to alleviate ER stress (e.g., sunitinib, approved for renal cell carcinoma, or tauroursodeoxycholic acid, approved for primary biliary cirrhosis) ( Involvement of autophagy impairment in proteinuria-induced tubular cell apoptosis and tubular injury Similarly to ER stress, autophagy can lead to either cell survival or death depending on the degree and type of stress

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