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Chronic -radiation-induced senescence and histone reduction rely on TP53 but not ATM To characterise the signalling mechanisms underpinning our observations, we examined the potential role of the tumour suppressor TP53 (p53) due to its key roles in regulating transcription and senescence in response to DNA damage 53,54

Crickets remained the subject of his scientific interest

59 Shafei reported that BCAT1 inhibited the Ras/ERK pathway and activates PI3K/AKT pathway through insulin/IGF-1, ultimately promoting the expression levels of FOXO3a and Nrf2 and regulating the proliferation, migration, and invasion in triple-negative breast cancer (TNBC)

The company itself also funded some of the research, creating an obvious potential bias

PMID 15598662