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Our findings align with previous studies with radical trapping antioxidants, i.e., ferrostatin-1 and liproxstatin-1, in MASLD models

While neither agent is specific for ferroptosis, these pre-clinical model studies suggest that a role for ferroptosis in the pathophysiology of IBD is worthy of greater consideration [37]

Research on the role of macrophages in myocarditis has focused primarily on the M1 and M2 polarization phenotypes, with a general consensus that M1 macrophages exacerbate myocarditis, whereas M2 macrophages alleviate myocardial inflammation

Introduction The ability to manage body oxygen stores in the face of environmental hypoxia constrains the life history of many vertebrate taxa