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Prasad AS, Bao B, Beck FW, Kucuk O, Sarkar FH

It was proposed that these cardioprotective effects are due to its inhibition of pro-hypertrophic signaling molecules, improvement of myocardial calcium handling, and reduction of oxidative stress and inflammation (Salehi et al., 2018)

doi: 10.3390/biom11030440

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This indicated that the autophagy level of T-BPH rat prostates was relatively elevated and could be further aggravated by ferroptosis inducer (RSL3), while the up-regulation of GPX3 expression (TRO treatment) could significantly antagonize autophagy induced by T or RSL3