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In H82, DKFZ-682-induced cell death was not mitigated by PARP or caspase inhibitors (olaparib or Z-VAD), suggesting these pathways are not involved (Fig

Percentages of proliferative (BrdU + ) (n = 6 technical replicates representative of 2 independent experiments) ( a ) and apoptotic (Annexin V + ) (n = 3 technical replicates representative of 3 independent experiments, **p = 0.0042) ( b ) cells were determined

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The main research takeaway is that Epithalon may interact with mitochondrial and oxidative-stress pathways in cell-culture conditions