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Ferroptosis as a therapeutic target in CVD Targeting the core pathological drivers of ferroptosisincluding dysregulated iron metabolism, uncontrolled lipid peroxidation, impairment of the GPX4 antioxidant axis, and additional regulatory pathways such as FSP1CoQ 10 , GCH1BH4, and DHODHrepresents a promising strategy to suppress ferroptosis in cardiovascular disease

When glucose was scarce, the levels of ROS were elevated (Fig

The dorsal putamen was selected as the center of the volume of interest due to its relatively homogenous mix of neurons and astrocytes, and suitable distance from bone and other regions that could compromise signal quality

Identification of the cyclamate interaction site within the transmembrane domain of the human sweet taste receptor subunit T1R3

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