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Therefore, we hypothesize that BPC157 may reduce intracellular ROS levels through an antioxidant mechanism similar to that reported in J Clin Invest [19], thereby stabilizing BACH1 protein and enabling its pro-angiogenic function

This data is consistent with other reports showing that the NO system response acts as an anti-inflammatory agent through inhibition of apoptosis on cells

dissolves readily in PBS and culture media Primary research origin: Watowich group, University of Texas at Austin Mechanism: competitive NNMT inhibition

Garbay, B., A.M