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found that the ALDH7A1 mutation is also present in folinic acid-responsive epilepsy, and combined treatment with PN and folinic acid may be considered for patients who do not respond to PN or folinic acid alone [33]

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Therefore, the crosstalk between LPO products such as 4-HNE and -catenin suggests that oxidative stress and lipid peroxidation are biphasic agents that can either contribute to pathological progression in, for example, normal tissues, or exert tumour -suppressive effects by masking oncogenic signals