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toxic metabolite formation or reduced glutathione stores, the reactive metabolite is free to covalently bind to vital proteins and the lipid bilayer of hepatocytes

Besides, pro-inflammatory mediators also hamper the production of brain-derived neurotrophic factor (BDNF), thus impairing BDNF signaling

Taken together, the effects of CCR2/CCR5 inhibition may improve some aspects of ASCVD

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