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Chi et al., 2011) (Table 3)

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This article is part of a review series focused on the role of cellular stress in driving molecular crosstalk between hepatic cells that may contribute to the development, progression, or pathogenesis of liver diseases

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Torres M, Jimenez S, Sanchez-Varo R et al (2012) Defective lysosomal proteolysis and axonal transport are early pathogenic events that worsen with age leading to increased APP metabolism and synaptic Abeta in transgenic APP/PS1 hippocampus

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frequently studied for recovery and muscle damage markers

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