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The data presented here suggest that Nlel-AnglV and Dihexa increase miniature synaptic activity by increasing dendritic spine densities and altering the morphological phenotype of postsynaptic spines in- vitro and may represent the mechanism that underlies facilitated learning observed AnglV analogues

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As stated above, the systemic treatment armamentarium in sarcoidosis is limited, while the likelihood of treatment failure is reported to be fairly high ( Commonly, treatment failure is interpreted as a drug being unsuccessful in inducing disease resolution or eliciting disease control