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Description

This paper presents the high-resolution stucture of an active-state G-protein-coupled receptor in complex with a G-protein peptide

M., Sousa, L

doi: 10.1002/fsn3.3227, 46

The ZDHHC21T209S/T209S mouse model confirms that the mutation significantly increases neuronal excitotoxicity susceptibility and triggers synaptic dysfunction by enhancing FYN palmitoylation, leading to NMDAR2B overactivation and dysfunction and neuronal loss, ultimately resulting in cognitive deficits (168)