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The mechanisms of action of SSRIs are commonly known as follows: first, SSRIs can selectively inhibit SERT, inhibiting the reuptake of 5-HT in the synaptic cleft and thereby exerting pharmacological effects

Moreover, the breakdown of -hydroxybutyrate generates metabolic intermediates like acetyl-CoA, succinyl-CoA, and Nicotinamide adenine dinucleotide (NAD), which can influence various intracellular pathways that regulate cell growth [44].Accordingly, metabolic reprogramming of cancer cells drive them toward glycolysis in the presence and absence of oxygen, called the Warburg effect [45]

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