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Additionally, the coloaded catalase facilitated oxygen production in high reactive oxygen species environments, helping to alleviate oxidative stress and hypoxia while drug was released from the nanostructure in a controlled manner

is, in part, supported by the US-NSF Division of Biological Infrastructure (DBI) Award#: 2330043

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CRC-infiltrating Th17 cells induced the expression of G-CSF by CAF leading to the recruitment to the TME of CD11b + Gr1 + myeloid cells endowed with pro-angiogenic features, and thus promoting resistance to VEGF blockade therapy (137)

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