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Based on these reports and our xenograft results, excessive collagen production in endometriosis appears to require not only the differentiation of fibroblasts into myofibroblasts but also activation of the TGFB/SMAD signaling pathway in the differentiated myofibroblasts via HOXC8

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In addition, POR is involved in ML210-induced ferroptosis (61), and P53 can affect ferroptosis without GPX4 inhibition (62)