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The mechanism by which cilostazol, a phosphodiesterase type 3 inhibitor, improves claudication is unknown, but the medication has the following properties: antiplatelet activity, vasodilatory properties, and in vitro inhibition of vascular smooth muscle cells

CPT1 deficiency can be identified by newborn screening using MS/MS [7]

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The included study subjects were diagnosed with AGA and displayed a Hamilton-Norwood status [28] [29] of II to V for men and a Ludwig status of I to II for women [30]